VIF

abbr.

virion infectivity factor 病毒体感染因子；

HIV-1 Vif protein binds the editing enzyme APOBEC3G and induces its degradation.
Role of vif in replication of human immunodeficiency virus type 1 in CD4+ T lymphocytes
Vif is crucial for human immunodeficiency virus type 1 proviral DNA synthesis in infected cells.
HIV-1 Vif blocks the antiviral activity of APOBEC3G by impairing both its translation and intracellular stability.
Vif Overcomes the Innate Antiviral Activity of APOBEC3G by Promoting Its Degradation in the Ubiquitin-Proteasome Pathway *
Induction of Ubiquitination and Degradation of APOBEC3G by HIV1 Vif-Cul5-SCF
Genetic characterization of vif, vpr, and vpu sequences from long-term survivors of human immunodeficiency virus type 1 infection.
The antiretroviral enzyme APOBEC3G is degraded by the proteasome in response to HIV-1 Vif
Isolation of a human gene that inhibits HIV-1 infection and is suppressed by the viral Vif protein.
A second human antiretroviral factor, APOBEC3F, is suppressed by the HIV‐1 and HIV‐2 Vif proteins